Glucostatic theory of appetite control/Bibliography: Difference between revisions

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(New page: {{subpages}} Mobbs, C.V., et al., Impaired glucose signaling as a cause of obesity and the metabolic syndrome: the glucoadipostatic hypothesis. Physiol Behav, 2005. 85(1): p. 3-23. Borne...)
 
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<ref name="pmid16580091">{{cite journal| author=Solomon A, De Fanti BA, Martínez JA| title=Peripheral ghrelin participates in the glucostatic signaling mediated by the ventromedial and lateral hypothalamus neurons. | journal=Peptides | year= 2006 | volume= 27 | issue= 7 | pages= 1607-15 | pmid=16580091 | url=http://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=16580091 | doi=10.1016/j.peptides.2006.02.004 }} </ref>
Mobbs, C.V., et al., Impaired glucose signaling as a cause of obesity and the metabolic syndrome: the glucoadipostatic hypothesis. Physiol Behav, 2005. 85(1): p. 3-23.
 
Bornet, F.R., et al., Glycaemic response to foods: impact on satiety and long-term weight regulation. Appetite, 2007. 49(3): p. 535-53.
 
Even, P. and S. Nicolaidis, Short-term control of feeding: limitation of the glucostatic theory. Brain Res Bull, 1986. 17(5): p. 621-6.
 
Stunkard, A., Satiety is a conditioned reflex. Psychosom Med, 1975. 37(5): p. 383-7.
 
Bernstein, L.M. and M.I. Grossman, An experimental test of the glucostatic theory of regulation of food intake. J Clin Invest, 1956. 35(6): p. 627-33.
 
Solomon, A., B.A. De Fanti, and J.A. Martinez, Peripheral ghrelin participates in the glucostatic signaling mediated by the ventromedial and lateral hypothalamus neurons. Peptides, 2006. 27(7): p. 1607-15.
[[User:Laura Sinclair|Laura Sinclair]] 13:14, 9 October 2010 (UTC)

Revision as of 07:18, 9 October 2010

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  1. ↑ Solomon A, De Fanti BA, Martínez JA (2006). "Peripheral ghrelin participates in the glucostatic signaling mediated by the ventromedial and lateral hypothalamus neurons.". Peptides 27 (7): 1607-15. DOI:10.1016/j.peptides.2006.02.004. PMID 16580091. Research Blogging.
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