Diabesity/Bibliography: Difference between revisions

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==Review Articles==
==Review Articles==


#Magni P. ''et al.'' (2009) Feeding behavior in mammals including humans. ''Ann.N.Y.Acad.Sci.'' 1163:221-232. PMID 19456343
#Magni P ''et al.''(2009) Feeding behavior in mammals including humans ''Ann NY Acad Sci'' 1163:221-32. PMID 19456343


Speakman J ''et al.''(2008) Thrifty vs Drifty Gene Theory of Obesity Thrifty genes for obesity, an attractive but flawed idea, and an alternative perspective: the 'drifty gene' hypothesis ''Int J Obesity''  32:1611–7
The discussion of thrifty genes and drifty genes, on obesity and diabetes. Suggestion made that the case for Thrifty genes may not be correct, but rather a different hypothesis is proposed
Cameron N ''et al.'' (2006) ''Childhood Obesity Contemporary Issues'' p154, CRC press, Taylor and Francis Group. (Discusses how obesity relates very closely to Diabetes. Relates genetic and environmental factors and summarises the thrifty gene hypothesis.)
Marchand L (2002) The Pima Indians: Pathfinders for health, Obesity and Diabetes, Online resource: [http://diabetes.niddk.nih.gov/DM/pubs/pima/obesity/obesity.htm]
NIDDK research conducted on the Pima Indians for the past 30 years has helped scientists prove that obesity is a major risk factor in the development of diabetes. One-half of adult Pima Indians have diabetes and 95% of those with diabetes are overweight. Scientists use the "thrifty gene" theory proposed in 1962 by geneticist James Neel to help explain why many Pima Indians are overweight
Neel JV (1962) Diabetes mellitus: a "thrifty" genotype rendered detrimental by "progress"? ''Am J Hum Genet'' 14:353–62. PMID 13937884. (The original paper that proposes the Thrifty genotype).
Again evidence that suggests the Thrifty genotype in Type 2 diabetes. And obesity
Joffe, Barry; Paul Zimmet (1998-08-17). The thrifty genotype in type 2 diabetes ''Endocrine'' 9:139–41. doi:10.1385/ENDO:9:2:139.
Zimmet P ''et al.'' (2001) Global and societal implications of the diabetes epidemic. Nature 414:782–787
Langendonk JG ''et al.'' (2006) Decrease in visceral fat following diet-induced weight loss in upper body compared to lower body obese premenopausal women ''Eur J Intern Med'' 17:465–9
Palaniappan L ''et al.''(2004) Predictors of the incident metabolic syndrome in adults. ''Diabetes Care'' 27:788–93
Nieves DJ ''et al.'' (2003) The atherogenic lipoprotein profile associated with obesity and insulin resistance is largely attributable to intra-abdominal fat. ''Diabetes'' 52:172–179.
Turner NC, Clapham JC (1998) Insulin resistance, impaired glucose tolerance and non-insulin dependent diabetes, pathologic mechanism and treatment: current status and therapeutic possibility. ''Drug Res'' 51:36–94
Gabriely I ''et al.'' (2002) Removal of visceral fat prevents insulin resistance and glucose intolerance of aging. ''Diabetes'' 51:2951–8
Tamara S ''et al.'' (2005) Childhood obesity and type 2 diabetes mellitus. ''Pediatrics'' 116:473-80
Garber J (2009). Restaging insulin therapy for patients with type 2 diabetes. ''Diabetes Obes Metab'' 11:1–5
Diabetes Prevention Program Research Group, Reduction in the incidence of type 2 diabetes with lifestyle intervention or metformin, ''N Engl J. Med'' 346: (2002) 393–403
Bray G. Drug Treatment of Obesity. Reviews in Endocrine & Metabolic Disorders. 2001; 2.
Lean MEJ ''et al.'' (1990) Obesity, weight loss and prognosis in type 2 diabetes ''Diabetic Med'' 7:228-33
Hartz AJ ''et al.'' (1983) Relationship of obesity to diabetes: influences of obesity level and body fat distribution. ''Prev Med'' 12:351-57
Umut Özcan ''et al.'' (2004) Endoplasmic Reticulum Stress Links Obesity, Insulin Action, and Type 2 Diabetes. ''Science'' 306:457-61


JOHN PARK BIBLIOGRAPHY (WILL SORT THIS OUT SOON)
Solinas G ''et al.''(2007) JNK1 in Hematopoietically Derived Cells Contributes to Diet-Induced Inflammation and Insulin Resistance without Affecting Obesity. ''Cell Metabol'' 6:386–97


1) Speakman J. et al. (2008) Thrifty vs Drifty Gene Theory of Obesity Thrifty genes for obesity, an attractive but flawed idea, and an alternative perspective: the 'drifty gene' hypothesis. International Journal of Obesity (2008) 32, 1611–1617
Karasawaa H ''et al.'' (2009) A novel model of type 2 diabetes mellitus based on obesity induced by high-fat diet in BDF1 mice. ''Metabolism Clinical and Experimental'' 58:296–303
The discussion of thrifty genes and drifty genes, on obesity and diabetes. Suggestion made that the case for Thrifty genes may not be correct, but rather a different hypothesis is proposed


2. Cameron, N et al. (2006) Childhood Obesity Contemporary Issues, p154, CRC press, Taylor and Francis Group.
Butler A ''et al.''(2003) β-Cell Deficit and Increased β-Cell Apoptosis in Humans With Type 2 Diabetes. ''Diabetes'' 52:102-10
Discusses how obesity relates very closely to Diabetes.
Relates genetic and environmental factors and summarises the thrifty gene hypothesis .


3. Marchand L, (2002) The Pima Indians: Pathfinders for health, Obesity and Diabetes, Online resource: http://diabetes.niddk.nih.gov/DM/pubs/pima/obesity/obesity.htm
Donath M ''et al.''(2008) Islet inflammation in type 2 diabetes: from metabolic stress to therapy. ''Diabetes Care'' 31:161-4
NIDDK research conducted on the Pima Indians for the past 30 years has helped scientists prove that obesity is a major risk factor in the development of diabetes. One-half of adult Pima Indians have diabetes and 95% of those with diabetes are overweight. Scientists use the "thrifty gene" theory proposed in 1962 by geneticist James Neel to help explain why many Pima Indians are overweight


4. Neel JV (1962). "Diabetes mellitus: a "thrifty" genotype rendered detrimental by "progress"?". Am. J. Hum. Genet. 14: 353–62. PMID 13937884.
Yogev Y, Visser G (2009) Obesity, gestational diabetes and pregnancy outcome. ''Seminars in Fetal and Neonatal Medicine'' 14:77-84


The original paper that proposes the Thrifty genotype.
Hattersley AT, Tooke JE (1999) The fetal insulin hypothesis: an alternative explanation of the association of low bir thweight with diabetes and vascular disease. ''Lancet''353:1789-92


Again evidence that suggests the Thrifty genotype in Type 2 diabetes. And obesity
Chu SY ''et al.''(2007) Maternal Obesity and Risk of Gestational Diabetes Mellitus. ''Diabetes Care'' 30:2070-6
Joffe, Barry; Paul Zimmet (1998-08-17). "The thrifty genotype in type 2 diabetes". Endocrine 9 (2): 139–141. doi:10.1385/ENDO:9:2:139.


==Primary Research Papers==
Boney CM ''et al.'' (2005) Metabolic syndrome in childhood: association with birth weight, maternal obesity, and gestational diabetes mellitus. ''Pediatrics'' 115


1) Umut Özcan, et al. (2004) Endoplasmic Reticulum Stress Links Obesity, Insulin Action, and Type 2 Diabetes. ''Science'' '''306''', 457-461.
http://www.webmd.com/baby/guide/pregnancy-gestational-diabetes
Through the use of cell culture and mouse models, this article shows that obesity causes stress in cells' endoplasmic reticulum. This leads to suppression of insulin receptor signalling via the hyper-activation of c-Jun N-terminal kinase (JNK) along with serine phosphorylation of insulin receptor substrate–1 (IRS-1). Mice which lack X- box–binding protein–1 (XBP-1), a transcription factor that modulates ER stress response, develop insulin resistance. The article argues that ER stress is an important feature of peripheral insulin resistance and type 2 diabetes at multiple levels. It is hypothesised that pharmacologic alteration of this pathway may offer chances of treating obesity.


2) Giovanni Solinas, et al. (2007) JNK1 in Hematopoietically Derived Cells Contributes to Diet-Induced Inflammation and Insulin Resistance without Affecting Obesity. ''Cell Metabolism''  '''6''', 386–397.
Dunger D, Ong K(2009) Diabetes in pregnancy: a vicious circle? ''Research Horizons'' Issue 8.
Obesity-induced insulin resistance is a major factor in the subsequent diagnosis of type 2 diabetes. Jun kinases (JNKs) are key negative regulators of insulin sensitivity in obese persons. Activation of JNKs (mainly JNK1) in insulin target cells causes the phosphorylation of insulin receptor substrates (IRSs) at serine and threonine residues that inhibit insulin signaling. JNK1 activation is also required for accumulation of visceral fat. Here we used reciprocal adoptive transfer experiments to determine whether JNK1 in myeloid cells, such as macrophages, also contributes to insulin resistance and central adiposity. Our results show that deletion of Jnk1 in the nonhematopoietic compartment protects mice from high-fat diet (HFD)-induced insulin resistance, in part through decreased adiposity. Conversely, removing JNK1 from hematopoietic cells has no effect on adiposity but causes protection against insulin resistance induced by a high fat diet, by decreasing obesity-induced inflammation.

Latest revision as of 07:49, 11 October 2010

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Review Articles

  1. Magni P et al.(2009) Feeding behavior in mammals including humans Ann NY Acad Sci 1163:221-32. PMID 19456343

Speakman J et al.(2008) Thrifty vs Drifty Gene Theory of Obesity Thrifty genes for obesity, an attractive but flawed idea, and an alternative perspective: the 'drifty gene' hypothesis Int J Obesity 32:1611–7 The discussion of thrifty genes and drifty genes, on obesity and diabetes. Suggestion made that the case for Thrifty genes may not be correct, but rather a different hypothesis is proposed

Cameron N et al. (2006) Childhood Obesity Contemporary Issues p154, CRC press, Taylor and Francis Group. (Discusses how obesity relates very closely to Diabetes. Relates genetic and environmental factors and summarises the thrifty gene hypothesis.)

Marchand L (2002) The Pima Indians: Pathfinders for health, Obesity and Diabetes, Online resource: [1] NIDDK research conducted on the Pima Indians for the past 30 years has helped scientists prove that obesity is a major risk factor in the development of diabetes. One-half of adult Pima Indians have diabetes and 95% of those with diabetes are overweight. Scientists use the "thrifty gene" theory proposed in 1962 by geneticist James Neel to help explain why many Pima Indians are overweight

Neel JV (1962) Diabetes mellitus: a "thrifty" genotype rendered detrimental by "progress"? Am J Hum Genet 14:353–62. PMID 13937884. (The original paper that proposes the Thrifty genotype).

Again evidence that suggests the Thrifty genotype in Type 2 diabetes. And obesity Joffe, Barry; Paul Zimmet (1998-08-17). The thrifty genotype in type 2 diabetes Endocrine 9:139–41. doi:10.1385/ENDO:9:2:139.

Zimmet P et al. (2001) Global and societal implications of the diabetes epidemic. Nature 414:782–787

Langendonk JG et al. (2006) Decrease in visceral fat following diet-induced weight loss in upper body compared to lower body obese premenopausal women Eur J Intern Med 17:465–9

Palaniappan L et al.(2004) Predictors of the incident metabolic syndrome in adults. Diabetes Care 27:788–93

Nieves DJ et al. (2003) The atherogenic lipoprotein profile associated with obesity and insulin resistance is largely attributable to intra-abdominal fat. Diabetes 52:172–179.

Turner NC, Clapham JC (1998) Insulin resistance, impaired glucose tolerance and non-insulin dependent diabetes, pathologic mechanism and treatment: current status and therapeutic possibility. Drug Res 51:36–94

Gabriely I et al. (2002) Removal of visceral fat prevents insulin resistance and glucose intolerance of aging. Diabetes 51:2951–8


Tamara S et al. (2005) Childhood obesity and type 2 diabetes mellitus. Pediatrics 116:473-80

Garber J (2009). Restaging insulin therapy for patients with type 2 diabetes. Diabetes Obes Metab 11:1–5

Diabetes Prevention Program Research Group, Reduction in the incidence of type 2 diabetes with lifestyle intervention or metformin, N Engl J. Med 346: (2002) 393–403

Bray G. Drug Treatment of Obesity. Reviews in Endocrine & Metabolic Disorders. 2001; 2.

Lean MEJ et al. (1990) Obesity, weight loss and prognosis in type 2 diabetes Diabetic Med 7:228-33

Hartz AJ et al. (1983) Relationship of obesity to diabetes: influences of obesity level and body fat distribution. Prev Med 12:351-57

Umut Özcan et al. (2004) Endoplasmic Reticulum Stress Links Obesity, Insulin Action, and Type 2 Diabetes. Science 306:457-61

Solinas G et al.(2007) JNK1 in Hematopoietically Derived Cells Contributes to Diet-Induced Inflammation and Insulin Resistance without Affecting Obesity. Cell Metabol 6:386–97

Karasawaa H et al. (2009) A novel model of type 2 diabetes mellitus based on obesity induced by high-fat diet in BDF1 mice. Metabolism Clinical and Experimental 58:296–303

Butler A et al.(2003) β-Cell Deficit and Increased β-Cell Apoptosis in Humans With Type 2 Diabetes. Diabetes 52:102-10

Donath M et al.(2008) Islet inflammation in type 2 diabetes: from metabolic stress to therapy. Diabetes Care 31:161-4

Yogev Y, Visser G (2009) Obesity, gestational diabetes and pregnancy outcome. Seminars in Fetal and Neonatal Medicine 14:77-84

Hattersley AT, Tooke JE (1999) The fetal insulin hypothesis: an alternative explanation of the association of low bir thweight with diabetes and vascular disease. Lancet353:1789-92

Chu SY et al.(2007) Maternal Obesity and Risk of Gestational Diabetes Mellitus. Diabetes Care 30:2070-6

Boney CM et al. (2005) Metabolic syndrome in childhood: association with birth weight, maternal obesity, and gestational diabetes mellitus. Pediatrics 115

http://www.webmd.com/baby/guide/pregnancy-gestational-diabetes

Dunger D, Ong K(2009) Diabetes in pregnancy: a vicious circle? Research Horizons Issue 8.

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