Metabolic acidosis/Bibliography: Difference between revisions
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* Halperin ML, Kamel KS. (2010) [http://dx.doi.org/10.1681/ASN.2009080794 Some observations on the clinical approach to metabolic acidosis]. ''J Am.Soc.Nephrol.'' 21(6):894-897. PMID 20488948. | * Halperin ML, Kamel KS. (2010) [http://dx.doi.org/10.1681/ASN.2009080794 Some observations on the clinical approach to metabolic acidosis]. ''J Am.Soc.Nephrol.'' 21(6):894-897. PMID 20488948. | ||
** The traditional clinical analysis for the presence of metabolic acidosis suffers from a number of limitations that at times hinders one's ability to reach a proper diagnosis. Our aim here is to raise awareness of some of the nuanced difficulties and illustrate why other considerations add value. | ** The traditional clinical analysis for the presence of metabolic acidosis suffers from a number of limitations that at times hinders one's ability to reach a proper diagnosis. Our aim here is to raise awareness of some of the nuanced difficulties and illustrate why other considerations add value. | ||
* Adrogue NE, Madias NE. (2010) [http://dx.doi.org/10.1681/ASN.2009121211 Secondary responses to altered acid-base status: the rules of engagement]. ''J Am.Soc.Nephrol.'' 21(6):290-923. PMID 20431042. | |||
** Each of the four canonical acid-base disorders expresses as a primary change in carbon dioxide tension or plasma bicarbonate concentration followed by a secondary response in the countervailing variable. Quantified empirically, these secondary responses are directional and proportional to the primary changes, run a variable time course, and tend to minimize the impact on body acidity engendered by the primary changes. Absence of an appropriate secondary response denotes the coexistence of an additional acid-base disorder. Here we address the expected magnitude of the secondary response to each cardinal acid-base disorder in humans and offer caveats for judging the appropriateness of each secondary response. | |||
Revision as of 16:33, 17 June 2010
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Books
- Gennari FJ, Adrogue HJ, Galla JH, Madias NE. (editors) (2005) Acid-Base Disorders and Their Treatment. Taylor & Francis Group: Boca Raton. ISBN 9780824759155. 856 pages.
- Rose BD, Post TW. (2001) Clinical Physiology of Acid-Base and Electrolyte Disorders. 5th ed. McGraw-Hill, Medical Pub. Division: New York. ISBN 0071346821.
Book chapters
- DuBose TD Jr. (2008) Disorders of Acid-Base Balance. Volume 1. Chapter 14. Page 505. In: Brenner BM (editor) (2008) Brenner & Rector's The Kidney. Eighth Edition. 2 volumes. Elsevier Inc., Saunders: Philadelphia. ISBN 978-1-4160-3105-5 (2-vol-set).
- Bevensee MO, Boron WF. (2008) Control of Intracellular pH. Volume 2. Chater 51. Page 1429. In: Alpern RJ, Hebert SC, Seldin DW, Giebisch GH. (editors) (2008) Seldin and Giebisch's The Kidney: Physiology & Pathophysiology. 2 volumes. Elsevier Inc., Academic Press: Amsterdam. ISBN 9780120884896. 2871 pages.
- Oh MS, Carroll HJ. (2008) External Balance of Electrolytes and Acids and Alkali. Volume 1. Chapter 10. Page 275. In: Alpern RJ, Hebert SC, Seldin DW, Giebisch GH. (editors) (2008) Seldin and Giebisch's The Kidney: Physiology & Pathophysiology. 2 volumes. Elsevier Inc., Academic Press: Amsterdam. ISBN 9780120884896. 2871 pages.
- Krapf R, Seldin DW, Alpern RJ. (2008) Clinical Syndromes of Metabolic Acidosis. Volume 2. Chapter 59. Page 1667. In: Alpern RJ, Hebert SC, Seldin DW, Giebisch GH. (editors) (2008) Seldin and Giebisch's The Kidney: Physiology & Pathophysiology. 2 volumes. Elsevier Inc., Academic Press: Amsterdam. ISBN 9780120884896. 2871 pages.
- Sebastian A, Frassetto LA, Morris RC Jr. (2008) The Acid-Base Effects of the Contemporary Western Diet: An Evolutionary Perspective. Volume 2. Page 1621. In: Alpern RJ, Hebert SC, Seldin DW, Giebisch GH. (editors) (2008) Seldin and Giebisch's The Kidney: Physiology & Pathophysiology. 2 volumes. Elsevier Inc., Academic Press: Amsterdam. ISBN 9780120884896. 2871 pages.
Journal articles
- Halperin ML, Kamel KS. (2010) Some observations on the clinical approach to metabolic acidosis. J Am.Soc.Nephrol. 21(6):894-897. PMID 20488948.
- The traditional clinical analysis for the presence of metabolic acidosis suffers from a number of limitations that at times hinders one's ability to reach a proper diagnosis. Our aim here is to raise awareness of some of the nuanced difficulties and illustrate why other considerations add value.
- Adrogue NE, Madias NE. (2010) Secondary responses to altered acid-base status: the rules of engagement. J Am.Soc.Nephrol. 21(6):290-923. PMID 20431042.
- Each of the four canonical acid-base disorders expresses as a primary change in carbon dioxide tension or plasma bicarbonate concentration followed by a secondary response in the countervailing variable. Quantified empirically, these secondary responses are directional and proportional to the primary changes, run a variable time course, and tend to minimize the impact on body acidity engendered by the primary changes. Absence of an appropriate secondary response denotes the coexistence of an additional acid-base disorder. Here we address the expected magnitude of the secondary response to each cardinal acid-base disorder in humans and offer caveats for judging the appropriateness of each secondary response.